...表明,与肾缺血相关的肾小管间质损伤可作为一种特殊机制,参与原发性高血压的发病,即盐敏感性高血压(salt-sensitive hypertension,SSHT)的发生与肾小管间质损伤有关[2].
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Results1. Salt-sensitive hypertension and renal injury animal model was established successfully.
结果1.成功建立盐敏感高血压合并肾脏损害动物模型。
参考来源 - 氨氯地平和依普利酮联合应用在缓解盐敏感高血压所致肾脏损害中作用机制的研究·2,447,543篇论文数据，部分数据来源于NoteExpress
Conclusions the over-expression of AT1R protein and AT1R mRNA in the heart and kidney may be associated with the increase of blood pressure in the rats with salt-sensitive hypertension.
结论感觉神经损伤性盐敏感性高血压大鼠心、肾at1r表达升高，AT 1 R表达水平的差异可能与盐敏感性高血压的形成有关。
Objective To study the effects of low sodium diet on the concentration of endothelin in patients with salt sensitive hypertension and salt resistive hypertension.
目的通过对原发性高血压病患者钠盐摄入的控制了解低盐饮食对盐敏感高血压及盐抵抗高血压病患者血中内皮素浓度的影响。
It is accepted that essential hypertension is the one that is associated with multiple genes. Research shows that almost 60% of the hypertensive patients are salt-sensitive.
目前原发性高血压被认为是一种多基因疾病，而我国北方高血压患者中接近60%为盐敏感高血压。
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